With 5 signs, CT can also diagnose hyperearly cerebral infarction

Cerebral infarction is one of the most common ischemic diseases, the incidence of cerebrovascular diseases in the first place, its disability rate and mortality rate is also very high, early diagnosis, early treatment is very important to improve prognosis, reduce disability and mortalityct flat sweep is the main method of diagnosis of acute cerebrovascular disease, with the improvement of diagnostic technology, some signs are found, which is of great significance for the diagnosis and treatment of super-early cerebral infarction (within 6 hours of onset)1theof the arterial high-density (HMCAS) in the brain (HMCAS)HMCAS is an imaging sign of arterial infarction in the brain, which is usually shown within 30 minutes of the onset of the disease and is an ultra-early sign of cerebral infarctionCT shows that the arterial density of one or both of the brain is higher than that of adjacent brain tissue, and a long strip or subsection-like high density shadow that travels continuously to the outside side of the saddle can be seen at the level of the saddle poolcomprehensive related literature, the occurrence of HMCAS is related to these factors:thrombosis or embolism caused by, it is generally believed that the single-sided brain arterial density increase syllands, when the blood sticky, slow blood flow or atherosclerotic plaque internal bleeding, necrosis caused by blood clots, embolism embolism in the brain artery caused by cerebral infarction;increasein blood cell ratio in the blood vessels caused by an increase in arterial density in the brain, when the blood cell ratio increased to 43.5%, thehigh blood pressure, diabetic blood vessels easy to calcification, which causes the blood vessel wall density increasedwhich means that when CT diagnoses cerebral infarction, high-density signs of arteries in the brain are also likely to be falsely positivehowever, calcice and blood cell increase caused by increased increase in the brain arterial density signs are more common on both sides, and patients do not have neurological symptoms and signs, and blood clots caused by the corresponding neuro-positional signs, we can distinguish from this pointin short, even if the CT test found this sign can not blindly draw conclusions, we also look at the density of the base artery changes, and whether the patient has neurological symptomsIf the density of the base artery also increases, and the patient does not have neuro-positional signs, then it can only be judged as cerebral arteriosclerosisFigure 1 patient, male, 80 years old, sudden left limb movement disorder 1 hour hospital CT flat sweep the right side of the brain arterial high density signs (left), 20 hours after the review of the right temporal lobe see flaky low density terrier (right) figure 2 patients, female, 63 years old, sudden consciousness, right limb movement disorder 30 minutes hospitalization CT flat sweep left brain arterial high density signs (left), 1 day after the review of the left frontal lobe saw large areas of low-density terrier (right) 2 bean-shaped nuclear signs bean-like nuclear signs is also one of the characteristics of early changes in cerebral infarction, CT as a bean-like nuclear outline blurred or partial disappearance mainly because the back of the shell core by the bean-print artery and the island screen artery together blood supply, when the bean-print artery far from the brain artery blockage, can trigger the island section screen artery blood supply disorder, the shell core back appeared intracellular edema, which in turn caused the bean-like nucleus outline blurred Figure 3 within 6 hours of the onset of the disease, the left bean-shaped nuclear sign (left, arrow, CT value 24Hu), the incidence of 48 hours of the corresponding region terrier (right, CT value 18 to 20Hu) 3 island with island belt Signs are the most sensitive indicators in the early stages of cerebral infarction, CT is shown to be the whole island belt or the rear island cortical boundary is blurred, the former is due to the main blocking of the arterial M2 section of the brain, and the latter is due to the closure of the arterial branch of the brain in the island leaf, large area of cerebral infarction patients are more common Figure 4 within 6 hours of the onset of the disease, the right island band signs (left, arrow refers to, CT value 22 to 24 Hu), the incidence of 48 hours of the corresponding region terrier (right, CT value 12 to 16Hu) 4 low density change
early low density change is mainly due to the intubation caused by intracellular edema after the infarction, resulting in an increase in cerebral tissue moisture content, brain tissue X-line absorption value slightly reduced in general some scholars believe that this low density change is different from the low density change after 6 hours, the former is mainly caused by cerebral edema, and the latter is an imaging change of ischemic necrosis of cerebral nerve cells in the terrier region Figure 5 Within 6 hours of the onset of the disease, the left low density change (left, arrow refers to, CT value 23 to 26Hu), the incidence of 48 hours of the corresponding region terrier (right, CT value 19 to 21Hu) 5 swelling of the groin, disappearing compared to , the incidence of this sign is lower, and at the same time can also appear a mild shift of the middle-line structure The cause may be an increase in capillary permeability in the terrier region, which leads to angiogenesis cerebral edema The appearance of cerebral edema makes the swelling of the groin shallower or even disappear Figure 6 Within 6 hours of the onset of the disease, the right-hand groin swelling, disappearance (left, arrow, CT value 20 to 23Hu), 48 hours of the disease of the corresponding region terrier (right, CT value 11 to 18Hu) source: video forum