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The Dorian B. McGavern team at the National Institutes of Health found that different myelin-like cell responses mediate damage and repair after cerebrovascular damage.
the study was published online January 18, 2021 in nature-neuroscience, a leading international academic journal.
researchers observed that the successful in-vascular thrombosis of acute large vascular thrombosis was associated with cerebral edema and poor clinical prognosis in patients with hemorrhagic transformation.
to understand this process, the researchers developed a cerebrovascular injury model using transcranial ultrasound, which was able to assess space-time in and around myelin cells.
found that injury and repair responses vary depending on time and cell origin.
-resident small glial cells initially stabilize damaged blood vessels in a way that is dependent on the carcogens, followed by a large influx of bone marrow monocytes.
Preventing myelin cell collection for long periods of time with anti-adhesive molecular therapy can prevent severe edema, but by interfering with subsequent vascular repair by small glial cells (RAM) associated with pro-inflammatory monocytes and angiogenesis repair, neuron destruction and fibrosis can be promoted.
data show how different myelin cell reactions over time can control, exacerbate, and ultimately repair cerebrovascular damage.
that cerebrovascular damage can lead to severe edema and inflammation, adversely affecting human health.
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