Scientists have discovered a protective shield for the human body.

Scientists have found a way to control the body's autoimmune response to promote tissue repair.the findings, published in current biology, reveal a new network of protective factors that can protect cells from damage.researchers at the University of Bristol have found that patients undergoing surgery can benefit significantly by accelerating recovery time and reducing the risk of complications.when a tissue is damaged (whether by accident or by surgery), the body will quickly gather immune cells to the injured site, and then phagocytize and kill the invading pathogens by releasing toxic factors (such as unstable molecules containing reactive oxygen species, such as peroxides), so as to fight against infection.however, these fungicides are also highly toxic to host tissues and can disrupt the repair process.to counteract these harmful effects, repair tissues activate powerful protective mechanisms to "protect" themselves from harm.now, researchers from the Bristol School of Biochemistry, who study tissue repair, have mapped out the exact identification of these protective pathways and determined how to stimulate the process in the original tissue.Dr Helen weaver of the Bristol School of life sciences, who was the lead author of the study, explained: "in healthy individuals, injured tissues usually repair themselves quickly after damage.in a healed skin wound, the stress response is activated to recruit inflammatory cells, which in turn release a variety of bactericidal factors, including reactive oxygen species (ROS), to eliminate invasive pathogens."in this study, the researchers used translucent fruit flies to observe the process of wound repair and to track the behavior of recruited immune cells.in doing so, researchers have found a network of protective pathways that can protect tissues from inflammatory damage and make repair tissues more "resilient" to stress.the researchers also demonstrated that ectopic activation of these pathways further enhanced tissue protection, while inhibition of these pathways resulted in a significant delay in wound healing."now that we know their identity and how they are activated, we hope to develop ways to stimulate this protective mechanism in patients before elective surgery. Dr. Weaver said.this finding has a significant clinical relevance to patients, because the clinical activation of these cytoprotective pathways can also provide an exciting "pretreatment" method for patients' tissues before selective surgery.he added: "we are now discovering more 'resilience' pathways that help protect our tissues from stress, both at the site of injury and in other vulnerable organs that are often exposed to similar stressors.because we found that the activation pathway of the protective mechanism is the same as that of the inflammatory response, we believe that the elastic mechanism has evolved into a failure protection mechanism of tissue protection every time inflammation is triggered.”