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Necrotizing enterocolitis (NEC) is a devastating disease of premature infants, and its pathogenesis is not fully understood, although the activation of the Gram-negative bacterial receptor Toll-like receptor 4 (TLR4) on the intestinal epithelium plays a key role Role
The picture comes from the Internet
The Johns Hopkins University School of Medicine and Johns Hopkins Children’s Center Pediatric Surgery conceived that the loss of intestinal glial cells in the premature intestine could lead to dyskinesia, excessive TLR4 signaling, and NEC
Figure 1 Impaired gastrointestinal motility and loss of intestinal glial cells are early events in NEC
Therefore, the loss of intestinal glial cells may lead to NEC through intestinal dyskinesia and increased TLR4 activation, suggesting that intestinal glial cells can treat the disease
Figure 2 Lack of BDNF/TrkB activity is the basis of the pathogenesis of AD
(Wang et al.
references
[1] Kovler ML, Gonzalez Salazar AJ, Fulton WB, et al.
[2] Wang ZH, Xiang J, Liu X, et al.
