Gout actually hurts the kidneys?! How can I protect my kidneys?

When we think of gout, we usually think of "heart-wrenching arthritis" or "high uric acid"
.

But in medicine, in addition to joint pain and high uric acid, what is more frightening is that gout can also cause a variety of harms, such as ischemic heart disease, obesity, hyperlipidemia, high blood pressure, kidney failure and so on
.

Among them, gout hurts the most deeply, which is our kidneys
.

Data surveys show that the incidence of kidney stones in gout patients is 200 times higher than that of normal people, 35%~40%.

In addition, 17%~25% of gout patients die of kidney failure
.

Therefore, for gout patients, "bad kidneys" is really not alarmist
.

What kidney diseases does gout cause?

In general, kidney damage caused by gout patients is mainly manifested in these three forms:

● Chronic urate nephropathy

Urate crystals are deposited in the renal interstitium, resulting in chronic tubular interstitial nephritis
.

It is mainly manifested as mild renal area soreness, early excretion of proteinuria or microscopic hematuria, but it is generally intermittent, so it is easy to be ignored;

● Acute uric acid nephropathy

The level of uric acid in blood and urine rises sharply, and a large number of uric acid crystals are deposited in renal tubules, collecting ducts, etc.
, resulting in blockage of urine flow, manifested as oliguria;

● Uric acid urinary tract stones

20%~25% of patients with primary gout will have uric acid urinary tract stones
.
Small stones are sediment-like and excreted with urine, often asymptomatic; If the stone is large, renal colic and hematuria will occur, and if the stone is repeated, it will cause urinary tract obstruction and local injury, and it is easy to be co-infected
.

Leave high uric acid alone

The kidneys will be more and more injured

Gout is a crystal-related arthropathy caused by monosodium urate deposition, which is directly related to hyperuricemia caused by purine metabolism disorders and/or decreased uric acid excretion, and belongs to the category
of metabolic rheumatism.

Under normal circumstances, purines are converted into uric acid in the human body, and 70% of uric acid in the human body is excreted through urine
.

However, if blood uric acid rises more than the body's own ability to excrete uric acid, it can cause hyperuricemia and gout
.

If high uric acid is not controlled for a long time, a large amount of uric acid in the blood of gout patients meets electrolytes to form urate, which is continuously deposited into the kidneys, and kidney damage will occur, resulting in renal insufficiency, and even uremia, which is life-threatening in severe cases!

Simply put, hyperuricemia or gout that cannot be effectively treated is the root cause
of "kidney damage".
Early active lowering of uric acid can delay or even prevent organ damage
.

Tips for lowering uric acid, remember these 4 points!

1.
Appropriate exercise: do not exercise too vigorously, in addition to maintaining the regularity of exercise;

2.
Adjust diet: do not drink alcohol and drinks containing fructose, avoid eating animal offal, seafood, hot pot, fish soup, broth, etc.
;

3.
Drink more water: not less than 2000 ml per day, preferably plain water, light tea can also be used, which can promote urination and help uric acid excretion;

4
.
Adhere to scientific medication: seek medical attention on time and monitor regularly.

Finally, Dr.
Huang Minyue, I still want to remind everyone that before the first gout attack, urate crystals have been deposited in the body for an average of 5~7 years, so the ablation of urate crystals is not completed overnight
.

Uric acid lowering treatment is a long-term process, which requires adherence to medication, medical treatment on time, development of healthy and good living and eating habits, and maintenance treatment with the minimum effective dose of drugs after uric acid reaches the standard
.
This is the best way
to "protect the kidneys".