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February 26, 2021 //---In patients with severe COVID-19, the innate immune system often overreacts.
This overreaction may be caused by thrombus formation and decreased blood oxygen saturation.
A study published by Uppsala University in Frontiers in Immunology shows this.
This overreaction may be caused by thrombus formation and decreased blood oxygen saturation.
A study published by Uppsala University in Frontiers in Immunology shows this.
Blood contains many proteins, which constitute the body's main barrier by identifying and destroying microorganisms, including SARS-CoV-2 (the virus that causes COVID-19).
These proteins are part of the intravascular innate immune system (IIIS), which consists of certain white blood cells, platelets and the so-called blood cascade system.
These proteins are part of the intravascular innate immune system (IIIS), which consists of certain white blood cells, platelets and the so-called blood cascade system.
(Image source: NIH)
At present, only 5% of animals have an immune system that includes T cells and B cells, while the remaining animals rely only on the natural immune system, which is mainly composed of IIIS.
With its inherent ability to recognize and eliminate foreign bodies and particles such as microorganisms and damaged cells, IIIS can be used as a "waste disposal system.
"
With its inherent ability to recognize and eliminate foreign bodies and particles such as microorganisms and damaged cells, IIIS can be used as a "waste disposal system.
"
In this study, researchers conducted a study on 66 severe COVID-19 hospitalized patients receiving care in the intensive care unit in 2020 and found that the activation of IIIS was obvious.
Professor Bo Nilsson of the Department of Immunology said: "This activation may be triggered by tissue damage and dead cells in the lungs.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
In terms of prognosis, the degree of activation is related to survival rate and lung function.
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
If IIIS plays a role that scientists suspect, it may be possible to treat severe COVID-19 symptoms with drugs that have been approved for the treatment of hereditary angioedema.
(Bioon.
com)
(Bioon.
com)
Information source: com/news/2021-02-innate-immune-worsens-situation-severe.
html">Innate immune system worsens the situation in severe COVID-19
html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
(Image source: NIH)
At present, only 5% of animals have an immune system that includes T cells and B cells, while the remaining animals rely only on the natural immune system, which is mainly composed of IIIS.
With its inherent ability to recognize and eliminate foreign bodies and particles such as microorganisms and damaged cells, IIIS can be used as a "waste disposal system.
"
With its inherent ability to recognize and eliminate foreign bodies and particles such as microorganisms and damaged cells, IIIS can be used as a "waste disposal system.
"
In this study, researchers conducted a study on 66 severe COVID-19 hospitalized patients receiving care in the intensive care unit in 2020 and found that the activation of IIIS was obvious.
Professor Bo Nilsson of the Department of Immunology said: "This activation may be triggered by tissue damage and dead cells in the lungs.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
In terms of prognosis, the degree of activation is related to survival rate and lung function.
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
If IIIS plays a role that scientists suspect, it may be possible to treat severe COVID-19 symptoms with drugs that have been approved for the treatment of hereditary angioedema.
(Bioon.
com)
(Bioon.
com)
Information source: com/news/2021-02-innate-immune-worsens-situation-severe.
html">Innate immune system worsens the situation in severe COVID-19
html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
In this study, researchers conducted a study on 66 severe COVID-19 hospitalized patients receiving care in the intensive care unit in 2020 and found that the activation of IIIS was obvious.
Professor Bo Nilsson of the Department of Immunology said: "This activation may be triggered by tissue damage and dead cells in the lungs.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
In terms of prognosis, the degree of activation is related to survival rate and lung function.
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
If IIIS plays a role that scientists suspect, it may be possible to treat severe COVID-19 symptoms with drugs that have been approved for the treatment of hereditary angioedema.
(Bioon.
com)
(Bioon.
com)
Information source: com/news/2021-02-innate-immune-worsens-situation-severe.
html">Innate immune system worsens the situation in severe COVID-19
html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
Professor Bo Nilsson of the Department of Immunology said: "This activation may be triggered by tissue damage and dead cells in the lungs.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
Due to increased vascular leakage, it may lead to clot formation and decreased blood oxygen saturation.
" It’s genetics and pathology.
In terms of prognosis, the degree of activation is related to survival rate and lung function.
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
If IIIS plays a role that scientists suspect, it may be possible to treat severe COVID-19 symptoms with drugs that have been approved for the treatment of hereditary angioedema.
(Bioon.
com)
(Bioon.
com)
Information source: com/news/2021-02-innate-immune-worsens-situation-severe.
html">Innate immune system worsens the situation in severe COVID-19
html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
In terms of prognosis, the degree of activation is related to survival rate and lung function.
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
Therefore, the new findings support the following view: IIIS is one of the driving factors of severe COVID-19.
In some patients with COVID-19, one explanation for why the IIIS works in this way may be that the cell damage is so great that the IIIS overreacts instead of helping to clear the tissue, making the situation worse .
If IIIS plays a role that scientists suspect, it may be possible to treat severe COVID-19 symptoms with drugs that have been approved for the treatment of hereditary angioedema.
(Bioon.
com)
(Bioon.
com)
Information source: com/news/2021-02-innate-immune-worsens-situation-severe.
html">Innate immune system worsens the situation in severe COVID-19
html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
If IIIS plays a role that scientists suspect, it may be possible to treat severe COVID-19 symptoms with drugs that have been approved for the treatment of hereditary angioedema.
(Bioon.
com)
(Bioon.
com)
Information source: com/news/2021-02-innate-immune-worsens-situation-severe.
html">Innate immune system worsens the situation in severe COVID-19
html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
Information source: com/news/2021-02-innate-immune-worsens-situation-severe.
html">Innate immune system worsens the situation in severe COVID-19
Information source: com/news/2021-02-innate-immune-worsens-situation-severe. html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
html">Innate immune system worsens the situation in severe COVID-19
Original source: Miklós Lipcsey et al.
com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
Original Source: com/news/2021-02-innate-immune-worsens-situation-severe. com/news/2021-02-innate-immune-worsens-situation-severe.
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System , Frontiers in Immunology (2021).
DOI: 10.
3389/fimmu.
2021.
627579
html">The Outcome of Critically Ill COVID-19 Patients Is Linked to Thromboinflammation Dominated by the Kallikrein/Kinin System Frontiers in Immunology
