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Posterior circulation ischemia (PCI) is a clinicopathological condition related to vertebrobasilar artery system infarction, mainly including the brain stem (48%) and the area dominated by the posterior inferior cerebellar artery (PICA) (36%).
PCI accounts for approximately 20-25% of all ischemic strokes, with an annual corrected incidence rate of 18/100,000 people.
Due to the large area of brain tissue that supplies blood to the vertebrobasilar system, PCI usually causes a variety of signs and symptoms, and rarely causes only one symptom.
The most common signs are unilateral limb weakness, facial paralysis, gait ataxia, dysarthria, and nystagmus.
Common symptoms are dizziness/dizziness, nausea and vomiting, headache and changes in consciousness.
Yimaitong compiles and organizes, please do not reprint without authorization.
PC can be subdivided into proximal, middle and distal intracranial regions.
The mid-segment area includes the brain area dominated by BA up to the pons dominated by its SCA branch and the cerebellum dominated by AICA.
This article continues to sort out the clinical manifestations of the mid-segment posterior circulation syndrome, as shown in Table 1.
Recommended reading↓↓↓Clinical manifestations of proximal intracranial circulation syndrome Table 1 Middle intracranial retrocirculation syndrome Note: PPRF: Parapontine midbrain reticular structure Yimaitong compiled from: Posterior circulation ischaemic stroke-a review part I: anatomy, aetiology and clinical presentations.
Neurol Sci.
2019 Oct;40(10):1995-2006.