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Posterior circulation ischemia (PCI) is a clinicopathological condition related to vertebrobasilar artery system infarction, mainly including the brain stem (48%) and the area dominated by the posterior inferior cerebellar artery (PICA) (36%).
PCI accounts for approximately 20-25% of all ischemic strokes, with an annual corrected incidence rate of 18/100,000 people.
Due to the large area of brain tissue that supplies blood to the vertebrobasilar system, PCI usually causes a variety of signs and symptoms, and rarely causes only one symptom.
The most common signs are unilateral limb weakness, facial paralysis, gait ataxia, dysarthria, and nystagmus.
Common symptoms are dizziness/dizziness, nausea and vomiting, headache and changes in consciousness.
Yimaitong compiles and organizes, please do not reprint without authorization.
Recommended reading↓↓↓Clinical manifestations of proximal intracranial circulation syndrome The clinical manifestations of mid-section intracranial retrocirculation syndrome PC can be subdivided into proximal, middle and distal intracranial areas.
The distal region includes the midbrain and thalamus dominated by BA, SCA, PCA and their perforators, and the cerebellum and PCA regions dominated by SCA.
This article continues to organize the clinical manifestations of the distal intracranial circulation syndrome, see Table 1.
Table 1 Distal intracranial posterior circulation syndrome Yimaitong compiled from: Posterior circulation ischaemic stroke-a review part I: anatomy, aetiology and clinical presentations.
Neurol Sci.
2019 Oct;40(10):1995-2006.