-
Categories
-
Pharmaceutical Intermediates
-
Active Pharmaceutical Ingredients
-
Food Additives
- Industrial Coatings
- Agrochemicals
- Dyes and Pigments
- Surfactant
- Flavors and Fragrances
- Chemical Reagents
- Catalyst and Auxiliary
- Natural Products
- Inorganic Chemistry
-
Organic Chemistry
-
Biochemical Engineering
- Analytical Chemistry
-
Cosmetic Ingredient
- Water Treatment Chemical
-
Pharmaceutical Intermediates
Promotion
ECHEMI Mall
Wholesale
Weekly Price
Exhibition
News
-
Trade Service
Guide
.
The immune response in AE is usually diffuse, leading to multifocal inflammation of the brain, and occasionally to the meninges, spinal cord, and/or peripheral nervous system
.
This diffuse inflammation may or may not be detected on adjunctive testing, but usually leads to multiple syndromes, which are a clinical feature
of AE.
Although some antibodies are associated with typical symptoms (e.
g.
, oral and mandibular dyskinesia in NMDAR antibody encephalitis, cognitive/behavioral changes, language and autonomic dysfunction, episodes of facial dystonia in LGI1 antibody encephalitis, etc.
), there is a clear symptom overlap
between all antibodies and all forms of AE.
Symptoms vary depending on the anatomical site of inflammation, and there are several clinical-anatomical syndrome categories in AE, as shown in
Table 1.
Table 2 AE clinical-anatomical syndrome categories
In 2019, Xiaobian launched the "Knowledge Card" column, which organizes some clinical disease-related knowledge points in the form of simple and easy-to-save tables, so that clinicians can use fragmented time to learn from the past, which is quite popular with doctor readers! This year, Xiaobian will continue to sort out the clinical knowledge points in the form of a table in order to help
clinicians.
.
The immune response in AE is usually diffuse, leading to multifocal inflammation of the brain, and occasionally to the meninges, spinal cord, and/or peripheral nervous system
.
This diffuse inflammation may or may not be detected on adjunctive testing, but usually leads to multiple syndromes, which are a clinical feature
of AE.
Although some antibodies are associated with typical symptoms (e.
g.
, oral and mandibular dyskinesia in NMDAR antibody encephalitis, cognitive/behavioral changes, language and autonomic dysfunction, episodes of facial dystonia in LGI1 antibody encephalitis, etc.
), there is a clear symptom overlap
between all antibodies and all forms of AE.
Symptoms vary depending on the anatomical site of inflammation, and there are several clinical-anatomical syndrome categories in AE, as shown in
Table 1.
Table 2 AE clinical-anatomical syndrome categories
Medical pulse through sorted from: medical pulse tong.
2 Figure 3 table teaches you the diagnosis of autoimmune encephalitis chart summary