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    Home > Active Ingredient News > Study of Nervous System > Bulbar infarction syndrome

    Bulbar infarction syndrome

    • Last Update: 2022-10-12
    • Source: Internet
    • Author: User
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    Bulbar infarction

    In the posterior circulation, atherosclerosis tends to occur proximal vertebral arteries (VA), distal intracranial segments of vertebral arteries, lower-middle BA, and proximal


    Clinical features

    Lateral dorsal syndrome (LMI)

    Dizziness and gait instability occur in more than 90% of patients, which may be due to


    Involvement of the suspected nucleus results in dysphagia, dysarthria, and hoarseness


    Sensory symptoms/signs are common, with only 4% of patients feeling normal


    Medullary medullary infarction syndrome (MMI)

    Dejerine proposed a triad of medial medullary infarction syndrome: contralateral central limb hemiplegia, without involvement of the facial nerve (pyramidal tract lesions); decreased or loss of contralateral deep sensation (medial thalamic damage); ipsilateral tongue muscle paralysis (damage to the sublingual nerves).


    Contralateral central limb hemiplegia without facial nerve involvement is the most typical clinical manifestation of MMI


    Sensory dysfunction is the second most important symptom/sign of MMI


    Vertigo/dizziness, nystagmus, and ocular movement disorders are strongly associated


    Significance of intracranial vertebral artery lesions in medulla oblongata syndrome

    Medulla oblongata is mainly supplied by some of the punctural arteries from the intracranial vertebral arteries (ICVAs


    Figure-1 88-year-old woman with a history of hypertension, clinical manifestations: dizziness and gait instability


    Wallenberg initially thought that PICA lesions were the cause of lateral dorsal infarction of


    For medullary ventral infarction syndrome, ASA occlusion was initially thought to be an important stroke mechanism


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