Activation of il33 protein to maintain insulin production in obese diabetic patients
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Last Update: 2017-11-24
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Source: Internet
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Author: User
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Now, the research results of diabetic inflammatory response have begun to show in clinical For example, the messenger material IL-1 - β is known to play a central role in the activation of the over morbid immune system Blocking IL-1 - β can inhibit diabetes and its cardiovascular complications Researchers from Basel University and affiliated hospitals found in a new study that an immune feedback mechanism helps maintain insulin production in overweight patients For ilc2 immune cells in the pancreas, the researchers found that under the condition of diabetes, the activated il33 protein can stimulate ilc2 cells to trigger obese individuals to release insulin using vitamin A acid, thus helping to alleviate the insulin production disorder This study adds new knowledge to the inflammatory network related to insulin production in diabetic patients, and further proves that the interaction between endocrine cells and immune cells is significant for maintaining insulin release.
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