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    Home > Active Ingredient News > Immunology News > The study revealed the molecular mechanism of soybean-subject kinase as a molecular switch to regulate disease-resistant immune triggers.

    The study revealed the molecular mechanism of soybean-subject kinase as a molecular switch to regulate disease-resistant immune triggers.

    • Last Update: 2020-10-06
    • Source: Internet
    • Author: User
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    Soybean is an important source of oil crops and plant proteins, but globally, the occurrence of persistent diseases (bacterial spot disease, plague root decay, soybean rust, etc.) has a serious impact on its yield and quality.
    plant's ability to fight pests and diseases is closely related to its own immune system, which consists of two main immune responses, microbial patterns that trigger immunity (pattern-triggered immunity, PTI) and effect factor-triggered immunity (effector-triggered immunity, ETI).
    current research on soybean disease-resistant molecular immune mechanism mainly focuses on ETI, while the research on PTI molecular mechanism is basically blank.
    The research team of Feng Xianzhong, a researcher at the Northeast Institute of Geography and Agricultural Ecology of the Chinese Academy of Sciences, and Dou Daolong, a professor at the Agricultural University of China, identified two allied gene mutations (Gmlmm1-1,2) through genetic screening of disease-like plaque mutants (lmms) associated with soybean autoimmunity, and the map clone confirmed that the gene encoded a Maletin-like subject kinase.
    GmLMM1 functionally missing mutants show cell death, PTI activation and reactive oxygen accumulation, enhanced ideotype resistance to bacterial and ovary pathogens, and after flog22 treatment, ROS bursts increased significantly, triggering an immune response to disease.
    further experiments show that GmLMM1 interacts with flg22 subject FLS2 and its co-subject BAK1, and negatively regulates the formation of a complex between the two induced by flg22.
    the above results confirm that GmLMM1 as a molecular switch to control moderate immune activation, GmLMM1 and PRR complex composition binding, negative regulation of PTI, so as to fine-tune the level of PTI, balanced immune response moderate occurrence.
    the study revealed the mechanism of identifying the microbial pattern triggered by the microbial pattern initiated by the pattern involved in GmLMM1 in soybeans, revealed the mechanism of soybean PTI disease resistance, and provided theoretical guidance for the cultivation of new varieties of highly disease-resistant soybeans.
    recently, the study was published in EMBO Reports under the title A Malectin-Like Receptor Kinase Regulates Cell Cell Death And Pattern-Triggered Immunity in Soy.
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