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Cerebral amyloid vascular disease (CAA) is one of intracerebral hemorrhage (ICH) and vascular dementia is the main reason for the world
Blood vessel
For many years, hemorrhagic MRI markers in the cerebellum were not considered a sign of CAA
This indicates that the accumulation of amyloid-β is not limited to the epithelial blood vessels, but also occurs in the cerebellar arteries that supply the superficial structures of the cerebellum
A distinction is usually made between (supermeningeal) cSS and submeningeal arachnoidopathy
They included 50 patients with Dutch-type hereditary CAA (average age 50 years), 45 patients with sporadic CAA (average age 72 years), and 43 patients with intracerebral hemorrhage associated with deep perforating arterial lesions (average age 54 years)
5 out of 50 Dutch type hereditary CAA patients (10%[95%CI, 2-18]) had superficial cerebellar spider web disease, 4/45 (9%[95%CI, 1-17]) In sporadic CAA patients, 0 (0%[95%CI, 0-8]) of 43 patients with intracerebral hemorrhage related to deep perforating arterial lesions were present
Compared with patients without this marker, patients with superficial cerebellar arachnoidopathy have more upper lobe and superficial cerebellar hemorrhage
The core conclusion of this study: superficial iron deposition in the cerebellum may be a new marker of CAA
Superficial cerebellar iron deposition may be a new marker of CAA
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