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Researchers at Ochsner Health and Tulane University School of Medicine have identified genes that become active in the carotid arteries when plaque ruptures leading to strok.
Cardiovascular disease is the leading cause of death worldwide, accounting for approximately 19 million deaths each year, usually as a result of plaque buildup in the arterie.
Previous studies relied on carotid artery samples taken months after a patient died or after a stroke or heart attac.
The researchers found that plaque rupture increases the inflammatory response and also leads to the loss of the part that protects the plaque from rupture, the fibrous ca.
Blockages in the carotid arteries, a common cause of some ischemic strokes, occur when blood supply to parts of the brain is interrupted, preventing brain tissue from getting necessary oxygen and nutrient.
"Inflammation is a known risk factor for atherosclerosis, which leads to stroke and heart attack," said Hernan Bazan, MD, the John Ochsner Professor of Cardiovascular Innovation at Ochsner Healt.
"Identifying the genes that promote this thinning gives us new therapeutic and diagnostic targets to prevent heart disease and stroke," emphasized the study's senior author, D.
The research was supported by the National Institutes of Health and the John Ochsner Professorship in Cardiovascular Innovatio.
