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68-year-old male patient with acute left myelin lateral infarction (pictured) with high-carbonated respiratory failure during repeated sleep during hospitalizationWhen awake, the patient is well ventilated, but when he sleeps, the patient develops severe respiratory acidosis (pH 7.14, pCO107), which becomes difficult to wake upPatients are given trachea intubation, and patient awareness improves rapidlyEventually, the patient had a tracheotomy and continued to experience prolonged sleep apnea during spontaneous breathtestsPrestroke patients do not have central sleep apneabreathing is divided into autonomy, marginality, automation (unconscious) and reflexivityTwo centers in the extended myelin control automatic respiration: the back-side respiratory center and the elitist mesh structureA single-sided myelin-back lesions can cause automatic respiratory cessation, which is caused by a cross of myelin spinal pathway damage leading to low deventilationcyatic central alveoli or Ondine cur syndrome(Figure: DWI can see the outside of the left eternity back and cerebellum infarction, in line with the lower arterial stroke of the cerebellum
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