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Neurology-new mechanism: why patients with migraine are prone to dizziness

 Last Update: 2021-07-27
 Source: Internet
 Author: User

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Migraine sufferers show enhanced vision and motor perception, including motion sickness and vestibular symptoms

diagnosis

Recently, modern visual self-motion stimulation based on a simulated roller coaster can study motion and gravity processing, motion sickness, and brain physiological mechanisms in an fMRI environment

Main hypothesis: Compared with subjects without migraine, migraine patients respond differently to visual stimuli in terms of clinical symptoms and brain processing mechanisms, especially in the thalamus-cerebellum-brainstem network

In addition, brain activity may be related to the phenotype of migraine, such as the presence/absence of vestibular symptoms, frequency of auras, and/or frequency of attacks

The neuronal activity of migraine patients is in the upper and lower occipital gyrus, pontine nucleus and cerebellar V/VI lobules, while the activity of cerebellar VIIb lobules and middle frontal gyrus is reduced

It was further found that in migraine patients, the connectivity between the cerebellar nucleus, cerebellar V/VI area, medial and supraoccipital gyrus and many cortical areas was enhanced, but not in the control group

In migraine patients, the connectivity between the cerebellar nucleus, cerebellar V/VI area, medial and supraoccipital gyrus and many cortical areas was enhanced, but not in the control group

It has been found that migraine is related to the abnormal regulation of visual motor stimulation in the superior and inferior occipital gyrus, middle frontal gyrus, pontine nucleus, cerebellar V, VI and VIIb lobules
.
It has been found that migraine is related to the abnormal regulation of visual motor stimulation in the superior and inferior occipital gyrus, middle frontal gyrus, pontine nucleus, cerebellar V, VI and VIIb lobules
.

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