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Nature Sub-Journal: LINC00842 promotes the malignant progression of pancreatic cancer through metabolic remodeling

 Last Update: 2021-09-06
 Source: Internet
 Author: User

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The molecular mechanism of pancreatic ductal adenocarcinoma (PDAC) is unclear

Pancreatic ductal carcinoma is one of the most deadly malignant tumors, with a 5-year survival rate of <9%

Recent studies have shown that many oncogenes and mutated tumor suppressor genes, such as c-myc and p53, are involved in regulating tumor cell metabolism

Long intergenic non-coding RNAs (LincRNAs) are a type of biological process regulators, which have been shown to play a role in metabolic regulation

Peroxisome proliferator-activated receptor gamma co-activator 1-α (pPARGC1A or pgc-1α) is the main transcriptional co-regulator involved in metabolic remodeling

In this study, the authors identified a metabolic remodeling ncRNA LINC00842 in PDAC, which can be induced by high glucose, which is a risk factor for the occurrence of PDAC

references

Xudong Huang et al.

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