Epigenetics Accelerates Aging, May Increase Risk of Colorectal Cancer

eLife [IF:14] ① This Mendelian randomization study assessed the association of epigenetic clocks measuring biological aging (HannumAge, Horvath, PhenoAge and GrimAge) with the risk of multiple cancers;② Obtained from meta-analyses and UK Biobank, FinnGen and the International Union for Genetics in Cancer Genome-wide association data of biological aging found limited evidence that accelerated epigenetic age is associated with breast, lung, ovarian, or prostate cancer;③Grimage accelerated age may increase the risk of colorectal cancer, compared with rectal cancer, The risk of colon cancer is greater;④ The risk of a variety of cancers can be reduced through lifestyle changes or epigenetic targeted thera. Assessing the causal role of epigenetic clocks in the development of multiple cancers: a Mendelian randomization study03-29, doi: 17554/eLi. 75374 Linked to a variety of adverse health outcomes, including canc. Epigenetics can be used to predict cancer risk, but whether epigenetic accelerated aging is associated with various cancer risks remains to be explor. Recently, eLife published a study comparing four established epigenetic clocks used to measure biological aging in association with the risk of multiple cancers through Mendelian randomization and found that epigenetic age acceleration is associated with breast cancer, breast cancer, Results of associations with lung, ovarian, e. have been inconsistent, but accelerated Grimage age may increase the risk of colorectal canc. In conclusion, this study provides new ideas for reducing cancer risk through lifestyle changes or clinical interventions in the futu.   Disclaimer: This article only represents the author's personal opinion and has nothing to do with China Probiotics Netwo. Its originality and the text and content stated in the text have not been verified by this site, and this site does not make any guarantee or commitment to the authenticity, completeness and timeliness of this text and all or part of its content and te. Readers are only for reference and please Verify the relevant content yourse. Copyright Notice Some articles reproduced on this site are not original, and their copyright and responsibility belong to the original auth. All reprinted articles, links and pictures on this website are for the purpose of conveying more information, and the source and author are clearly indicat. Media or individuals who do not wish to be reprinted can contact us for infringing information that can provide sufficient eviden. , bio149 will be deleted within 12 hours after confirmati. Users are welcome to submit original articles to 86371366@. com, which will be published on the homepage after review, and the copyright and responsibility of the articles belong to the send.

eLife① This Mendelian randomization study assessed the association of epigenetic clocks measuring biological aging (HannumAge, Horvath, PhenoAge and GrimAge) with the risk of multiple cancers;② Obtained from meta-analyses and UK Biobank, FinnGen and the International Union for Genetics in Cancer Genome-wide association data of biological aging found limited evidence that accelerated epigenetic age is associated with breast, lung, ovarian, or prostate cancer;③Grimage accelerated age may increase the risk of colorectal cancer, compared with rectal cancer, The risk of colon cancer is greater;④ The risk of a variety of cancers can be reduced through lifestyle changes or epigenetic targeted thera.
Assessing the causal role of epigenetic clocks in the development of multiple cancers: a Mendelian randomization study03-29, doi: 17554/eLi.
75374 Linked to a variety of adverse health outcomes, including canc.
Epigenetics can be used to predict cancer risk, but whether epigenetic accelerated aging is associated with various cancer risks remains to be explor.
Recently, eLife published a study comparing four established epigenetic clocks used to measure biological aging in association with the risk of multiple cancers through Mendelian randomization and found that epigenetic age acceleration is associated with breast cancer, breast cancer, Results of associations with lung, ovarian, e.
have been inconsistent, but accelerated Grimage age may increase the risk of colorectal canc.
In conclusion, this study provides new ideas for reducing cancer risk through lifestyle changes or clinical interventions in the futu.

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eLife① This Mendelian randomization study assessed the association of epigenetic clocks measuring biological aging (HannumAge, Horvath, PhenoAge and GrimAge) with the risk of multiple cancers;② Obtained from meta-analyses and UK Biobank, FinnGen and the International Union for Genetics in Cancer Genome-wide association data of biological aging found limited evidence that accelerated epigenetic age is associated with breast, lung, ovarian, or prostate cancer;③Grimage accelerated age may increase the risk of colorectal cancer, compared with rectal cancer, The risk of colon cancer is greater;④ The risk of a variety of cancers can be reduced through lifestyle changes or epigenetic targeted thera.

Assessing the causal role of epigenetic clocks in the development of multiple cancers: a Mendelian randomization study03-29, doi: 17554/eLi.

75374 Linked to a variety of adverse health outcomes, including canc.

Epigenetics can be used to predict cancer risk, but whether epigenetic accelerated aging is associated with various cancer risks remains to be explor.

Recently, eLife published a study comparing four established epigenetic clocks used to measure biological aging in association with the risk of multiple cancers through Mendelian randomization and found that epigenetic age acceleration is associated with breast cancer, breast cancer, Results of associations with lung, ovarian, e.

have been inconsistent, but accelerated Grimage age may increase the risk of colorectal canc.

In conclusion, this study provides new ideas for reducing cancer risk through lifestyle changes or clinical interventions in the futu.

---

eLife[IF:14]① This Mendelian randomization study assessed the association of epigenetic clocks (HannumAge, Horvath, PhenoAge and GrimAge) measuring biological aging with risk of multiple cancers;② From a meta-analysis and from UK Biobank, FinnGen and The International Union of Genetics on Cancer obtained genome-wide association data of biological aging and found that there is limited evidence that epigenetic age acceleration is associated with breast, lung, ovarian or prostate cancer;③ Grimage age acceleration may increase the risk of colorectal cancer, and Compared with rectal cancer, the risk of colon cancer is higher;④ The risk of various cancers can be reduced by lifestyle changes or epigenetic targeted thera.

Assessing the causal role of epigenetic clocks in the development of multiple cancers: a Mendelian randomization study03-29, doi: 17554/eLi.

75374 Linked to a variety of adverse health outcomes, including canc.

Epigenetics can be used to predict cancer risk, but whether epigenetic accelerated aging is associated with various cancer risks remains to be explor.

Recently, eLife published a study comparing four established epigenetic clocks used to measure biological aging in association with the risk of multiple cancers through Mendelian randomization and found that epigenetic age acceleration is associated with breast cancer, breast cancer, Results of associations with lung, ovarian, e.

have been inconsistent, but accelerated Grimage age may increase the risk of colorectal canc.

In conclusion, this study provides new ideas for reducing cancer risk through lifestyle changes or clinical interventions in the futu.

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