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Hypertrophic cell hyperplactivity is a blood system tumor characterized by clone amplification of KIT D816V mutant hypertrophic cells in multiple organs, leading to severe and even life-threatening allergic reactions.
Recent studies have found that hereditary alpha-trypsinemia (H-T) may be a common genetic feature of hypertrophic cell hypertrophic disorder, with an increase in the number of copies of the alpha-trypsin encoded gene TPSAB1, which is associated with elevated levels of the underlying serum trypsinase and an increased risk of hypertrophic cell activity.
the study was designed to shed light on the clinical rate of H alpha T in patients with hypertrophic cell hypertrophic disorder.
assessed the TPSAB1 embryonic copy number variation in 180 hypertrophic cell hypertrophic patients, 180 gender-matched control individuals, 720 other myelin tumor patients, and an additional 61 hypertrophic cell hypertrophic patients in a separate validation queue with PCR.
, H-alphaT-compatible TBSAB1 copy amplification was detected in 17.2% of hypertrophic patients and 4.4% of control individuals.
levels of trypsin in patients with H alphaT plus were higher than those in patients with H alphaT-patients (49.6 ng/mL vs. 34.5 ng/mL, p?0.004) and were independent of the fat cell load.
patients with H-T-hypertrophic cell hyperactive disorders were more likely to have hypersensitive reactions and severe cardiovascular media-related symptoms/allergic reactions than patients with H alpha T-hypertrophic cell hyperactive disorders.
the above results are confirmed in a separate validation queue.
summary, the high rate of H alphaT in hypertrophic cell hyperplate suggests that the number of copies of coded TPSAB1 amplification has a potential pathogenic effect in disease development.
H-alpha T is a new biomarker of hypertrophic cell hypertrophy that can be used to determine the risk of severe allergic reactions in a single individual.
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