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Existing studies have shown that particulate air pollution is an environmental risk factor for cognitive dysfunction in the elderly.
It also accelerates the loss of cognitive function, increases the risk of dementia, loss of gray matter and white matter in the brain, and small vessel disease
.
In rodent models, existing studies have documented the neurotoxicity of air pollution components, including cognitive impairment, oxidative damage and increased brain amyloid, as well as neurogenesis in adulthood
Signs of recent decline in neurotoxicity of air pollution components in human populations and rodent models of blood vessels Recent signs of decline in neurotoxicity of air pollution components in human populations and rodent models
In the results of the US-wide Health and Retirement Study (HRS), the cognitive deficits of the less-educated cohort in 2004 were strongly associated with air pollution: among adults 65 years of age and older, PM2.
5 With an increase of 5μg/m3 and education time ≤8 years, the risk of cognitive deficits will increase by 12%
.
However, by 2014, the corresponding HRS cohort had no such association
In adults 65 years of age and older, for every 5μg/m3 increase in PM2.
A possible factor is the reduction of PM2.
Studies in rodent models have shown that the neurotoxicity of air pollution from urban settings is reduced
.
The nano-level sub-components (nPM) of PM2.
In these observations, the components of air pollution are also changing
.
The PM2.
The composition of air pollution is also changing .
The protective effect of increased education on the risk of dementia caused by air pollution may be a factor leading to these results
Therefore, the researchers emphasized that the results of the study cannot assess the potential benefits of improved air pollution on cognitive decline and the risk of dementia
.
Although PM2.
Original source:
Original source:Jennifer A.
Jennifer A.
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